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Psychologist

Relapse Prevention for Addictive Behaviors

4.00 - CE credit hours training

$25.00 - Cost of course

This page contains important information about the course, including training details, a comprehensive course description, learning objectives, and source references. We encourage you to review each section to gain a clear understanding of the course content, educational goals, and supporting materials before beginning your training and taking the POST TEST below.

Target audience and instructional level of this course: foundational
There is no known conflict of interest or commercial support related to this CE program.

Relapse Prevention for Addictive Behaviors

Follow the link below, read the material and then take the 24 question post test

Read the Course Material:
Relapse Prevention for Addictive Behaviors

24 Questions

1. 1. From "Introduction": Relapse prevention is a critical focus, given that twelve month relapse rates following substance cessation attempts range from ____________.
2. From "Definitions of Relapse Prevention and Relapse": The authors view relapse as:
3. From "Marlatt's RP model": In Marlatt's relapse prevention model a lapse can be followed by a number of outcomes. When the problem behavior is corrected and the desired behavior is re-instantiated, this is referred to as a ______________.
4. From "Marlatt's RP model": The Abstinence Violation Effect:
5. From "the Reformulated CBT model": Which of the following would be considered a tonic process?
6. From "the Reformulated CBT model": In the reformulated CBT model ________ processes can determine who is vulnerable for relapse, while _________ processes determine when relapse occurs.
7. From "the Reformulated CBT model": The reformulated model remains grounded in cognitive behavioral theory, but adds:
8. From "Empirical Findings Relevant to the RP model": The meta-analysis of 26 RP treatment outcome studies found RP had a larger effect on:
9. From "Empirical Findings Relevant to the RP model": The Magill and Ray meta-analysis of CBT for substance use disorders found that _____ of those who received CBT had better outcomes than those in comparison groups.
10. From "Self-Efficacy": The authors describe the first study to examine relapse in relation to phasic and tonic self-efficacy levels. It found that individuals with low, medium and high baseline self-efficacy diverged on their momentary self-efficacy ratings:
11. From "Outcome Expectancies": Research on attentional bias modification (ABM) has shown that ABM:
12. From "Outcome Expectancies": Outcome expectancies are defined as anticipated outcomes of a given behavior or situation.
13. From "Withdrawal": The authors conclude that the core aspect of withdrawal that affects relapse motivation is:
14. From "Negative Affect": Overall, the cited studies on negative affect and substance use point to:
15. From "Self-control and coping responses": What does the existing evidence about coping responses and lapses/relapses say?
16. From "Using nonlinear methods to model relapse": Nonlinear models appear to better account for relapse drinking than linear models.
17. From "Genetic influences on treatment response and relapse": The authors cite three primary contexts in which genetic variation could influence liability for relapse during or following treatment. One context is genetic variations that could impact pharmaceutical treatment response. The authors cite a large Naltrexone outcome difference based on gene variation on the OPRM1 gene. Did this occur amongst patients receiving Naltrexone alone or Naltrexone plus CBT?
18. From "Genetic influences on treatment response and relapse": In Project Match, among those with the high-risk genotype, drinking behavior outcomes were similar regardless of treatment condition. However, treatment differences emerged in the low-risk genotype group, such that ____ produced the best outcomes, followed by ____.
19. From "Mindfulness-based RP": One mindfulness-based RP techniques teaches clients to view urges as analogous to an ocean wave that rises, crests, and diminishes. This is known as:
20. From "Mindfulness-based RP": In a randomized controlled study comparing MBRP to 12-step treatment as usual, MBRP participants reported:
21. From "Critiques of the RP Model": The NIAA study testing the validity of the original RP taxonomy generated supportive data.
22. From "Mechanisms of Treatment Effects": In CBT for substance use changes in coping skills, self-efficacy, and/or outcome expectancies are the primary mechanisms purported to have effects. What is the research evidence for these mechanisms?
23. From "Integrating implicit cognition and neurocognition in relapse models": In the dual process model of addiction controlled thoughts are known as _____________ and automatic thoughts are known as _______________.
24. From "Conclusions and Policy Implications": How does the use of RP fit into the view that addiction is a chronic condition?
The Relapse Prevention (RP) model has been a mainstay of addictions theory and treatment since its introduction three decades ago. This course provides an overview and update of RP for addictive behaviors with a focus on developments over the last decade (2000-2010). Major treatment outcome studies and meta-analyses are summarized, as are selected empirical findings relevant to the tenets of the RP model. Notable advances in RP in the last decade include the introduction of a reformulated cognitive-behavioral model of relapse and the development of mindfulness-based relapse prevention. Also reviewed is the emergent literature on genetic correlates of relapse following pharmacological and behavioral treatments.
  • Identify developments in relapse prevention from 2000-2010.
  • Summarize the impact of relapse prevention approaches on a variety of clinical factors.
  • Describe mindfulness-based relapse prevention.
  • Describe genetic and neurological influences on treatment response and relapse.
Christian S Hendershot, Ph.D.
Centre for Addiction and Mental Health
Department of Psychiatry, University of Toronto

Katie Witkiewitz, Ph.D.
Department of Psychology, Washington State University

William H George, Ph.D.
G Alan Marlatt, Ph.D.
Department of Psychology, University of Washington